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The conformational conversion of the cellular form of the prion protein (PrP C ) into the infectious form (PrP Sc ) and the proteolytic processing of the amyloid-β (Aβ) peptide are central pathogenetic events in the prion diseases and Alzheimer's disease, respectively. Cholesterol- and sphingolipid-rich lipid rafts have emerged as important sites for the conversion of PrP C ...
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