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Brain to blood transport is believed to be a major determinant of the amount of amyloid β protein (AβP) found in brain. Impaired efflux has been suggested as a mechanism by which AβP can accumulate in the CNS and so lead to Alzheimer's disease (AD). To date, however, no study of the efflux of the form of AβP most relevant to AD, AβP1-42, has been conducted, even though a single amino acid substitution...
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