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Traumatic brain injury is a major cause of death and disability worldwide and often associated with post-traumatic epilepsy. We recently demonstrated that TBI induces acquired GABA A receptors channelopathy that associates with hyperexcitability in granule cell layer (GCL). We now assessed the expression of GABA A and GABA B receptor subunit mRNAs between 6 h and 6 months post-TBI...
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