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Experimental evidences now indicate that memory formation relies on the capacity of neuronal networks to manage long-term changes in synaptic communication. This property is driven by N-methyl-d-aspartate receptors (NMDAR), which requires the binding of glutamate but also the presence of the co-agonist d-serine at the glycine site. Defective memory function and impaired brain synaptic plasticity observed...
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