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Over-expression of α7 nicotinic acetylcholine receptor (α7nAChR) in PC12 cells, independent of agonistic stimulation, induces marked neurite outgrowth and high capacity for migration and adherence (differentiation-like transformation), and increases tolerance against cell damage. In the present study, we investigated the effects of α7nAChR over-expression and nicotine on ERK phosphorylation and N-cadherin...
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