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Assemblyof the amyloid-β peptide (Aβ) into fibrils and its deposition in distinct brain areas is considered responsible for the pathogenesis of Alzheimer's disease (AD). Thus, inhibition of fibril assembly is a potential strategy for therapeutic intervention. Electron cryomicroscopy was used to monitor the initial, native assembly structure of Aβ42. In addition to the known fibrillar intermediates,...
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