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Abnormal Glu release occurs in the spinal cord of SOD1G93A mice, a transgenic animal model for human ALS. Here we studied the mechanisms underlying Glu release in spinal cord nerve terminals of SOD1G93A mice at a pre-symptomatic disease stage (30days) and found that the basal release of Glu was more elevated in SOD1G93A with respect to SOD1 mice, and that the surplus of release relies on synaptic...
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