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Mice deficient for the IL-1RAcP gene (IL-1RAcP KO) were used to explore the role of IL-1RAcP in physiological functions of brain IL-1β. Animals were injected i.c.v. with two different doses of recombinant human (rh) IL-1β: a small one (750 pg) known to induce sickness behavior, and a larger one (50 ng), chosen to counteract the possible loss of affinity of IL-1β on its receptor. Neuroendocrine and...
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