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Thiazolidinediones (TZDs) are insulin-sensitising drugs that are ligands for the nuclear receptor PPARγ. They have been shown to inhibit PMA-stimulated secretion of TNFα from human monocytes, although only at concentrations well in excess of circulating levels observed during TZD therapy, suggesting a mechanism of action independent of PPARγ activation. Here we show that insulin-sensitising concentrations...
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