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The mechanism involved in [Ca 2+ ] i -dependent feedback inhibition of store-operated Ca 2+ entry (SOCE) is not yet known. Expression of Ca 2+ -insensitive calmodulin (Mut-CaM) but not wild-type CaM increased SOCE and decreased its Ca 2+ -dependent inactivation. Expression of TrpC1 lacking C terminus aa 664-793 (TrpC1ΔC) also attenuated Ca...
In the present work, we studied the interaction and effect of several IP 3 receptor (IP 3 R) constructs on the gating of the store-operated (SOC) hTrp3 channel. Full-length IP 3 R coupled to silent hTrp3 channels in intact cells but did not activate them until stores were depleted of Ca 2+ . By contrast, constructs containing the IP 3 -binding domain activated...
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