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AbstractObjective. Reperfusion injury may affect the cardiac NO and endothelin production. We investigated whether 20 min of total ischemia followed by 40 min of reperfusion can induce apoptosis in a Langendorff model of retrogradely perfused rat hearts (37C; paced at 300/), and we attempted to correlate these findings with measured tissue NO and ET-1 levels. Methods. An apoptosis detection system...
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