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Zinc is an endogenous N‐methyl‐D‐aspartate (NMDA) receptor blocker. It is possible that zinc‐mediated modification of hippocampal CA1 long‐term potentiation (LTP) is linked to the expression of NMDA receptor subunits, which varies with postnatal development. In the present study, the effect of ZnCl2 and CaEDTA, a membrane‐impermeable zinc chelator, on CA1 LTP induction was examined in hippocampal...
The function of zinc released from the neuron terminals is poorly understood. Here, the action of zinc in excitatory neurotransmission in rat hippocampal CA1 was studied by using in vivo microdialysis. Glutamate concentration in the perfusate was significantly decreased by perfusion with 10-300μM ZnCl 2 , suggesting that presynaptic release of glutamate is inhibited by zinc in the CA1. While...
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