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Scope
The fetal programming in response to over‐nutrition during pregnancy is involved in pathogenesis of cardiovascular diseases later in life. The authors’ previous work reported that prenatal high‐sucrose (HS) diet impaired functions of large‐conductance Ca2+‐activated K+ channels (BK) in mesenteric arteries in the adolescent offspring rats. This study determines whether prenatal HS has a long‐term...
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