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Accumulation of the β‐amyloid (Aβ) peptide in extracellular senile plaques rich in copper and zinc is a defining pathological feature of Alzheimer′s disease (AD). The Aβ1–x (x=16/28/40/42) peptides have been the primary focus of CuII binding studies for more than 15 years; however, the N‐truncated Aβ4–42 peptide is a major Aβ isoform detected in both healthy and diseased brains, and it contains a...
Gehirnwäsche! Ein Platin‐Komplex (siehe Schema) wurde entwickelt, der oral gegeben werden kann und die Amyloid‐Belastung in den Gehirnen transgener Mausmodelle mit Alzheimer‐Krankheit reduziert. Analysen von Gehirngewebe zufolge führte die Behandlung mit der Pt‐Verbindung zu einer 26 %‐igen Verminderung der Zahl an Amyloid‐β‐Peptid‐Plaques.
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