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Double-strand breaks in mammalian DNA lead to rapid phosphorylation of C-terminal serines in histone H2AX (γ-H2AX) and formation of large nuclear γ-H2AX foci. After DNA repair these foci disappear, but molecular mechanism of elimination of γ-H2AX foci remains unclear. H2AX protein can be phosphorylated and dephosphorylated in vitro in the absence of chromatin. Here, we compared global exchange of...
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