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In this paper, we use a basal ganglia-corticothalamic model (BGCT) to study control effect of the absence epilepsy seizure. It is shown that the seizure state can be well controlled by tuning activation level of the thalamic reticular nucleus (TRN), specific relay nuclei (SRN), striatal D1 neurons and striatal D2 neurons. And then, one type of deep brain stimulation voltage employed on SRN, we find...
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