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Mitochondrial membrane potential (DYm) is one of the key drivers of free radical production. Changes in DYm strongly affect free radical turnover in both mitochondria and cytosol. We found that in cytochrome c oxidase deficient HCT116 cells F1Fo ATP synthase (mATPase) is reversed in order to maintain DYm polarisation. For small energy costs, mATPase enables mitochondrial protein import, Ca2+ turnover...
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