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In the mammalian pancreas, high concentrations of Zn 2+ are co-secreted with insulin, which may then permeate via abundant L-type Ca 2+ channels (LTCC) present on the β-cells. Neither the mechanisms utilized by these cells to lower cytosolic Zn 2+ nor the implications of increased intracellular Zn 2+ on β-cell survival are well understood. To address this, we employed...
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