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Decreased Na+/K+‐ATPase activity, and both sirtuin 1 (SIRT1) and adenosine monophosphate‐activated protein kinase (AMPK) have been reported to be involved in the development of diabetic cardiomyopathy (DCM).The present study aimed to investigate the advanced glycation end‐products (AGE) that impair Na+/K+‐ATPase stability by regulating the AMPK/SIRT1 pathway during progression of DCM.To study type...
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