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BackgroundThis study aimed to investigate the possible synergistic effects of lipid disorder with renin–angiotensin system (RAS) activation in non‐alcoholic fatty liver disease (NAFLD).
MethodsApolipoprotein E gene‐knockout mice, angiotensin II (Ang II) type 1 receptor (AT1) gene‐knockout mice and human hepatoblastoma cell line (HepG2) were used for experiments. Lipid accumulation was examined by...
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