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Mice lacking the tumor suppressor gene p53 spontaneously develop T‐cell lymphomas at a high rate, suggesting that in these mice lymphomas arise due to defective apoptosis mechanisms in T cells mediated by p53. However, a role of p53 in regulation of T‐cell responses or apoptosis has been poorly defined. TCR‐mediated signaling in the absence of CD28 costimulation induces both apoptosis and proliferation...
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