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Our previous study has confirmed that Lactobacillus acidophilus KLDS 1.0738 (La KLDS 1.0738) could alleviate β‐lactoglobulin (β‐Lg)‐induced allergic inflammation. This study further explored its molecular regulation mechanism through an in vitro macrophage model. β‐Lg‐induced macrophages were treated with strains of viable or non‐viable L. acidophilus and Toll‐like receptor 4 (TLR4) inhibitor or...
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