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Adenosine possesses potent anti‐inflammatory properties which are partly mediated by Gi‐coupled adenosine A3 receptors (A3Rs). A3R agonists have shown clinical benefit in a number of inflammatory conditions although some studies in A3R‐deficient mice suggest a pro‐inflammatory role. We hypothesised that, in addition to cell signalling effects, A3R compounds might inhibit neutrophil chemotaxis by disrupting...
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