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Aim
This study was designed to determine whether ET‐1 derived from endothelial cells contributes to oxidative stress in the glomerulus of mice subjected to a high‐salt diet and/or hypoxia.
Methods
C57BL6/J control mice or vascular endothelial cell ET‐1 knockout (VEET KO) mice were subjected to 3‐h exposure to hypoxia (8% O2) and/or 2 weeks of high‐salt diet (4% NaCl) prior to metabolic cage assessment...
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