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Background and Aims
Acetaminophen (APAP) overdose induces severe liver injury and hepatic failure. While the activation of c‐Jun NH2‐terminal kinase (JNK) has been implicated as a mechanism in APAP‐induced liver injury, the hepatic defense system controlled by nuclear factor erythroid 2–related factor 2 (Nrf2) plays a central role in the mitigation of APAP toxicity. However, the link between the...
Hepatocellular carcinoma (HCC) is a cancer lacking effective therapies. Several measures have been proposed to treat HCCs, such as senescence induction, mitotic inhibition, and cell death promotion. However, data from other cancers suggest that single use of these approaches may not be effective. Here, by genetic targeting of Survivin, an inhibitor of apoptosis protein (IAP) that plays dual roles...
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