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Background
Erectile function is usually impaired after radiation therapy in prostate cancer patients. eNOS is a key enzyme in the process of erection. Mitochondria‐associated membranes (MAMs) are closely contacted with the production and bioactivity of eNOS.
Objective
To study the mechanism of icariin improves the erectile function of rats treated with prostate radiation by controling the expression...
Objectives
To investigate the effect of icariin on endothelial microparticles, endothelial progenitor cells, platelets, and erectile function in spontaneously hypertensive rats.
Materials and methods
Twelve 8‐week‐old healthy male Wistar‐Kyoto rats and 12 spontaneously hypertensive rats were randomly divided into four following groups: Wistar‐Kyoto control group (normal saline 1 ml/d given by gavage),...
Background
Type 5 phosphodiesterase inhibitor (PDE5I) has become the first‐line treatment for erectile dysfunction (ED). However, its effective rate for hypertension ED is only 60%‐70%. How to improve the efficacy of ED treatment is the focus of current research.
Objective
To explore whether icariin can improve the erectile function of spontaneously hypertensive rats (SHR) by affecting post‐translational...
IntroductionThe impaired erectile response in spontaneously hypertensive rats (SHR) is caused by increased signaling of RhoA/Rho‐kinase and decreased signaling of nitric oxide (NO). Icariin improves erectile function via upregulating multitargets in NO/cyclic guanosine monophosphate (NO/cGMP) pathway, which breviscapine accomplishes by downregulating RhoA/Rho‐kinase pathway.
AimTo investigate the...
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