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Activation of the unfolded protein response in combination with generation of reactive oxygen species, from cytochrome P450 members and NADPH‐P450 reductases, are two major consequences of Endoplasmic Reticulum (ER) stress that cause oxidative damage and cell death. Herein, we reviewed the role of Bax Inhibitor‐1 (BI‐1), an evolutionarily conserved protein encoded by the Transmembrane Bax inhibitor...
Background Oxidative stress, inflammation, and endoplasmic reticulum (ER) stress play a major role in the pathogenesis of neonatal hypoxic-ischemic (HI) injury. ER stress results in the accumulation of unfolded proteins that trigger the NADPH-P450 reductase (NPR) and the microsomal monooxygenase system which is composed of cytochrome P450 members (CYP) generating reactive oxygen species (ROS) as well...
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