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Alzheimer's disease (AD) has a characteristic hallmark of amyloid-β (Aβ) accumulation in the brain. This accumulation of Aβ has been related to its faulty cerebral clearance. Indeed, preclinical studies that used mice to investigate Aβ clearance showed that efflux across blood–brain barrier (BBB) and brain degradation mediate efficient Aβ clearance. However, the contribution of each process to Aβ...
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