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The mechanisms altering knee flexion in hemiparetic gait may be neurological (muscle overactivity) or orthopedic (soft tissue contracture) in nature, a distinction which is difficult to ascertain clinically during gait. This study aimed to distinguish the 2 mechanisms in evaluating thigh–shank coordination, which may show instability across the gait cycle in the case of bursting rectus femoris overactivity...
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