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Aims
Neuroinflammation and pyroptosis are key mediators of cerebral ischemia/reperfusion (I/R) injury‐induced pathogenic cascades. BRCC3, the human homolog of BRCC36, is implicated in neurological disorders and plays a crucial role in neuroinflammation and pyroptosis. However, its effects and potential mechanisms in cerebral I/R injury in mice are unclear.
Methods
Cellular localization of BRCC3...