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A 17 kDa fragment of human growth hormone (22 kDa hGH), identified as hGH44–191, has lower binding affinity for growth hormone receptors (GHRs), but has been reported to be more potent in producing glucose intolerance in yellow obese mice. Our aim was to investigate this anomaly by comparing acute development of hyperinsulinemia and insulin resistance (“diabetogenic activity”) during hGH44–191 or...
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