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Simulated ischemic conditions (hypoxia-hypoglycaemia) in vitro enhanced glutamate efflux from rat cerebrocortical prisms. Here we characterised efflux mechanisms using pharmacological tools. The Na + channel blocker TTX (1 μM) did not affect ischemia-induced efflux, while sipatrigine (100 μM), a Na + /Ca 2+ channel blocker and ω-conotoxin MVIIC (2 μM), an N/P/Q type Ca...
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