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AimsGalectin‐1, a member of the β‐galactoside‐binding lectin family, accumulates in neurofilamentous lesions in the spinal cords of both sporadic and familial amyotrophic lateral sclerosis (ALS) patients with a superoxide dismutase 1 gene (SOD1) mutation (A4V). The aim of this study was to evaluate the roles of endogenous galectin‐1 in the pathogenesis of ALS.
MethodsExpression of galectin‐1 in the...
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