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The cellular mechanisms underlying the exceptional vulnerability of the basal forebrain (BF) cholinergic neurons during pathological aging have remained elusive. Here we employed an adeno‐associated viral vector‐based RNA interference (AAV‐RNAi) strategy to suppress the expression of tropomyosin‐related kinase A (trkA) receptors by cholinergic neurons in the nucleus basalis of Meynert/substantia innominata...
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