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The development of Alzheimer's disease (AD) is generally thought to correlate with cerebral accumulation of Aβ. It has previously been shown that Aβ peptides enhance vasoconstriction in isolated arteries and oppose certain vasorelaxants. Moreover, exogenous application of Aβ peptides causes cerebral vasoconstriction in rodents and in transgenic mouse models of AD that overexpress Aβ there is reduced...
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