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Stimulation of cells with tumor necrosis factor-α (TNF-α) results in the increase in generation of H 2 O 2 in mitochondria that leads to apoptosis. The effect of H 2 O 2 produced by TNF-α on the redox status of selenocysteine (SeCys) residue essential for mitochondrial thioredoxin reductase (TrxR2) was investigated in HeLa cells. TNF-α caused accumulation of oxidized...
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