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Objective Mutations of the gsp oncogene are responsible for 30–40% of GH‐producing pituitary adenomas and 10% of nonfunctioning pituitary adenomas (NFPAs). However, the pathogenetic mechanism of the remaining pituitary tumours still remains to be identified. Recently, the interaction between the chemokine stromal cell‐derived factor 1 and its receptor CXCR4 was found to play an important role in...
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