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Objective: Calcium entry via the L-type Ca 2+ channel (LTCC) is crucial for excitation-contraction (EC) coupling and activation of Ca 2+ -dependent signal transduction pathways in cardiac myocytes. Both nitric oxide (NO), signaling via cGMP, and acetylcholine, signaling via the muscarinic receptor, have been identified as negative regulators of β-adrenoreceptor-stimulated...
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