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The DNA damage response factor 53BP1 functions at the intersection of two major double strand break (DSB) repair pathways – promoting nonhomologous end-joining (NHEJ) and inhibiting homology-directed repair (HDR) – and integrates cellular inputs to ensure their timely execution in the proper cellular contexts. Recent work has revealed that 53BP1 controls 5′ end resection at DNA ends, mediates synapsis...
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