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The roles of cyclic AMP (cAMP)–ERK1/2–Bad signaling pathways in 6-hydroxydopamine (6-OHDA)-induced cell survival and death were investigated. In PC12 cells, 6-OHDA (10–100μM) concentration-dependently increased the intracellular levels of cAMP mediated by the Ca 2+ -CaMKII-adenylyl cyclase system. 6-OHDA at the non-toxic level (10μM) induced transient ERK1/2 phosphorylation and BadSer112 phosphorylation,...
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