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Both APP and PS-1 are causal genes for early-onset familial Alzheimer’s disease (AD) and their mutation effects on cerebral Aβ deposition in the senile plaques were examined in human brains of 29 familial AD (23 PS-1, 6 APP) cases and 14 sporadic AD cases in terms of Aβ40 and Aβ42. Aβ isoform data were evaluated using repeated measures analysis of variance which adjusted for within-subject measurement...
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