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Background: Transmissible spongiform encephalopathies are a group of neurodegenerative disorders of man and animals that are believed to be caused by an α-helical to β-sheet conformational change in the prion protein, PrP. Recently determined NMR structures of recombinant PrP (residues 121–231 and 90–231) have identified a short two-stranded anti-parallel β sheet in the normal cellular form of the...
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