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To investigate the potential involvement of adrenergic signaling in Alzheimer's disease (AD) pathogenesis, we performed genetic and functional studies of genes initiating the cascade. We chose two functional single-nucleotide polymorphisms (SNPs) in the β1-adrenergic receptor (ADRB1) and the G protein β3 subunit (GNB3) genes, respectively, and analyzed their allelic frequencies in a case-control sample...
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