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AbstractWe investigated the effects of a protein kinase A (PKA) inhibitor, H-89 {N-[2-(p-bromocinnamylamino)ethyl]-5-iso-quinolinesulphonamide}, on Ca2+ regulation in Fura-2-loaded ferret myocytes. H-89 (10 mol/l) decreased the amplitude of the Fura-2 transient to 28.24.3% (P0.001) of control and prolonged its duration, characterized by a decrease in the rate of decline of Ca2+ to diastolic levels:...
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