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Spontaneous autoimmune polyneuropathy (SAP) in B7‐2 knock‐out non‐obese diabetic (NOD) mice is mediated by myelin protein zero (P0)‐reactive T helper type 1 (Th1) cells. In this study, we investigated the role of B cells in SAP, focusing on CD19 as a potential therapeutic target. We found that P0‐specific plasmablasts and B cells were increased in spleens of SAP mice compared to wild‐type NOD mice...
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