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Recent data from several groups suggest that the primary mechanism of amyloid β-protein (Aβ) neurotoxicity may be mediated by free radicals. To evaluate this hypothesis, our aim is to make the mechanism of Aβ neurotoxicity clear, especially in the formation of free radicals. In this study, rat pheochromocytoma (PC12) cells were exposed to Aβ25–35 and confirmed free radical generations using two kinds...
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