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In the past decades, several cases of the syndrome of generalized glucocorticoid (GC) resistance have been reported. This familial disease is characterized by reduced cortisol effects, due to a GC receptor (GR) defect, which is compensated by hyperactivity of the hypothalamic–pituitary–adrenal (HPA) axis. As a consequence, patients present with signs of adrenal overproduction of mineralocorticoids...
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