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Inflammation is one of the major causes of intervertebral disc degeneration (IDD). Emerging evidence has revealed that increase in the levels of pro‐inflammatory cytokines, such as interleukin 6 (IL‐6) and tumor necrosis factor alpha (TNF‐α), can activate a variety of signaling pathways, eventually resulting in IDD. Here, we show that the two cullin family genes, CUL4A and CUL4B, but not other cullins,...
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