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The second heart sound (S2) is triggered by an aortic valve closure as a result of the ventricular-arterial interaction of the cardiovascular system. The objective of this paper is to investigate the timing of S2 in response to the changes in hemodynamic parameters and its relation to aortic blood pressure (BP). An improved model of the left ventricular-arterial interaction was proposed based on the...
The onset of second heart sound is triggered by the closure of aortic valve due to the interaction of left ventricle and arterial system. Noninvasive experiments found that RS2 defined by the time delay from the peak of ECG R wave to the onset of the second heart sound had a close inverse correlation with arterial systolic blood pressure. However, no theoretical study has been carried out to investigate...
The onset of second heart sound is triggered by the closure of aortic valve due to the interaction of left ventricle and arterial system. Noninvasive experiments found that RS2 defined by the time delay from the peak of ECG R wave to the onset of the second heart sound had a close inverse correlation with arterial systolic blood pressure. However, no theoretical study has been carried out to investigate...
Previous studies have shown that it is possible to estimate pulmonary and systemic blood pressure using the spectral information of the second heart sound. A mathematical model for the vibration of the closed aortic valve is proposed in the paper with the introduction of the nonlinear elasticity of the aortic wall tissue. Based on the parametric analysis, the effect of the aortic blood pressure at...
Previous studies have shown that it is possible to estimate pulmonary and systemic blood pressure using the spectral information of the second heart sound. A mathematical model for the vibration of the closed aortic valve is proposed in the paper with the introduction of the nonlinear elasticity of the aortic wall tissue. Based on the parametric analysis, the effect of the aortic blood pressure at...
The coronary artery disease is a major cause of deaths in the western world. One indicator for coronary artery disease (CAD) is coronary artery calcification (CAC). An accurate and reproducible scheme is desired to monitor the progression of patient's coronary calcification in follow-up studies. Traditional approaches for CAC estimation lack to provide accurate and reproducible results. In This work,...
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