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Neuroinflammation can be caused by various factors in early brain injury after subarachnoid hemorrhage (SAH). One of the most important features of this process is M1 microglial activation. In turn, the TLR4/NF‐κB pathway plays an essential role in activating M1 phenotypic microglia. Biglycan, a small leucine‐rich proteoglycan, functions as an endogenous ligand of TLR4 and TLR2 in macrophages. However,...
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